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Scientists have discovered how the brain gets toxic particles that contribute to Alzheimer’s disease. Lipoproteins may be responsible for this process. For now, the research is carried out on genetically modified mice. Human research is still a long way from now, but it is a lead that could explain the cause of this serious degenerative disease.
- Scientists around the world are working hard to discover the cause of Alzheimer’s disease
- One theory is the amyloid hypothesis, in which the beta-amyloid protein may be responsible for the development of the disease
- Australian scientists have found that toxic compounds can enter the brain via lipoproteins
- The research was published in the American scientific journal PLOS Biology
- You can find more such stories on the TvoiLokony home page
There are several hypotheses in research on the causes of Alzheimer’s disease, which are still inconclusive. Among them are also as amazing as the impact of the herpes simplex virus in causing disease in some people, oxidative stress and air pollution. One of the most advanced theories of Alzheimer’s disease, to which scientists devote a lot of research is amyloid hypothesis.
The amyloid hypothesis is one of the possible causes of the development of Alzheimer’s disease
For many years, scientists have suspected that a fragment of the beta-amyloid protein may be responsible for the development of Alzheimer’s disease. They study this particular chain of amino acids and its role in the body. They know that if it clumps in the brain and forms deposits, the prognosis for a patient’s health is not good. In recent years, the focus has been on how beta-amyloid travels throughout the body and brain and on the relationship of beta-amyloid with a type of fat-transfer chemical called lipoproteinami.
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It’s the lipoproteins that might be to blame
Using a transgenic mouse model, researchers at the Curtin Health Innovation Research Institute in Australia, an institute whose researchers focus on studying disease in the elderly, have found compelling evidence that beta-amyloid proteins produced elsewhere in the body are transferred to the brain via via the lipid transport system.
– Our research shows that these Toxic protein deposits that form in the brains of people living with Alzheimer’s disease most likely travel to the brain from fat-carrying molecules in the blood called lipoproteins – says lead researcher and physiologist John Mom. “This blood-brain pathway is important because, if we manage to control the levels of lipoprotein-amyloid in the blood and prevent them from leaking into the brain, it opens up potential new treatments to prevent Alzheimer’s disease and slow memory loss,” he adds.
As emphasized by the scientists in the research published in the American scientific journal PLOS Biology, there is still much to be done, but the discovery suggests that dietary treatments for Alzheimer’s disease may exist or the use of pharmaceuticals that manage the interaction of lipoproteins with beta-amyloid.
September 21 is the World Day for Combating Alzheimer’s Disease
About 400 people suffer from Alzheimer’s in Poland. people, around 50 million in the world. On September 21, we celebrate the World Alzheimer’s Disease Day. Dementia as a whole ailments, including Alzheimer’s disease, is considered one of the most serious health problems of the XNUMXst century.
Projections of the number of Alzheimer’s sufferers predict that due to the aging of the world population, the number of patients will increase in the coming decades.
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